Skip Navigation


Mol. Hum. Reprod. Advance Access originally published online on July 24, 2009
Molecular Human Reproduction 2009 15(10):577-586; doi:10.1093/molehr/gap057
This Article
Right arrow Full Text
Right arrow Full Text (PDF)
Right arrow All Versions of this Article:
15/10/577    most recent
gap057v1
Right arrow Alert me when this article is cited
Right arrow Alert me if a correction is posted
Services
Right arrow Email this article to a friend
Right arrow Similar articles in this journal
Right arrow Similar articles in PubMed
Right arrow Alert me to new issues of the journal
Right arrow Add to My Personal Archive
Right arrow Download to citation manager
Right arrowRequest Permissions
Google Scholar
Right arrow Articles by Braundmeier, A.G.
Right arrow Articles by Fazleabas, A.T.
PubMed
Right arrow PubMed Citation
Right arrow Articles by Braundmeier, A.G.
Right arrow Articles by Fazleabas, A.T.
Social Bookmarking
Add to CiteULike   Add to Connotea   Add to Del.icio.us  
What's this?

© The Author 2009. Published by Oxford University Press on behalf of the European Society of Human Reproduction and Embryology. All rights reserved. For Permissions, please email: journals.permissions@oxfordjournals.org

This article appears in the following Molecular Human Reproduction issue: Special Issue: Mechanisms of Endometriosis [View the issue table of contents]

The non-human primate model of endometriosis: research and implications for fecundity

A.G. Braundmeier and A.T. Fazleabas1

Department of Obstetrics and Gynecology (MC808), College of Medicine, University of Illinois at Chicago, 820 South Wood Street, Chicago, IL 60612, USA

1 Correspondence address. E-mail: asgi{at}uic.edu

The development of an animal model of endometriosis is crucial for the investigation of disease pathogenesis and therapeutic intervention. These models will enhance our ability to evaluate the causes for the subfertility associated with disease and provide a first-line validation of treatment modulators. Currently rodents and non-human primate models have been developed, but each model has their limitations. The aim of this manuscript is to summarize the current findings and theories on the development of endometriosis and disease progression and the effectiveness of therapeutic targets using the experimental induced model of endometriosis in the baboon (Papio anubis).

Key words: animal model/endometriosis/endometrium/primate/uterine/pathologies

Submitted on May 26, 2009; resubmitted on July 1, 2009; accepted on July 8, 2009.


Add to CiteULike CiteULike   Add to Connotea Connotea   Add to Del.icio.us Del.icio.us    What's this?




Disclaimer: Please note that abstracts for content published before 1996 were created through digital scanning and may therefore not exactly replicate the text of the original print issues. All efforts have been made to ensure accuracy, but the Publisher will not be held responsible for any remaining inaccuracies. If you require any further clarification, please contact our Customer Services Department.