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Mol. Hum. Reprod. Advance Access published online on May 29, 2009

Molecular Human Reproduction, doi:10.1093/molehr/gap037
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© The Author 2009. Published by Oxford University Press on behalf of the European Society of Human Reproduction and Embryology. All rights reserved. For Permissions, please email: journals.permissions@oxfordjournals.org

GRP78 as a marker of preeclampsia: an exploratory study

A. Laverrière1, R. Landau2, I. Charvet3, O. Irion1, P. Bischof1, M. Morales1 and M. Cohen1,a

1Department of Obstetrics and Gynaecology, Maternity, University of Geneva, Boulevard de la Cluse, 1211, Geneva 14, Switzerland 2Department of Anesthesiology, University of Washington Medical Center, Seattle, WA 98195-6540 3Department of Anesthesia, University Hospital of Geneva, Geneva, Switzerland

a To whom correspondence should be addressed: Marie Cohen, Laboratoire d'Hormonologie, 32 bd de la Cluse, 1211 Genève 14, Switzerland, Tel : (41) 22 38 24 381; Fax : (41) 22 38 24 310; e-mail : marie.cohen{at}hcuge.ch

Although the exact mechanisms that lead to shallow invasion or defective trophoblastic differentiation in preeclampsia are still unknown, it is widely admitted that the etiology of preeclampsia is a defect in trophoblast invasion of the uterine spiral arteries. We have previously observed that the status of a chaperone protein, glucose regulated protein 78 (GRP78) is associated with the invasive properties of cytotrophoblastic cells, we therefore hypothesized that circulating GRP78 could serve as a diagnostic tool in preeclampsia. In a prospective case-control study, we quantified GRP78 autoantibodies, complexes of GRP78 with autoantibodies, and GRP78 (C-term fragment, N-term fragment, and full-length GRP78) by ELISA. Plasma from women diagnosed with preeclampsia (n=16), from women during the first trimester of pregnancy who subsequently developed preeclampsia (n=10) and from healthy pregnant women (controls, n=58 at term, n=26 at first trimester) were analyzed and compared. We observed no significant difference between preeclamptic and healthy pregnant women for autoantibodies-GRP78 complexes or total GRP78 at both first trimester and at delivery. In contrast, the ratio of C-terminal GRP78 over full length GRP78 was significantly different in plasma of preeclamptic patients as compared to controls both during first trimester (p<0.004) and at term (p<0.0001). Our findings suggest that circulating C-terminal GRP78 reflect the invasive properties of cells, and could be used as a predictive marker for preeclampsia early in pregnancy.

Key Words: GRP78/C-terminal fragment/marker/preeclampsia

Submitted on November 19, 2008; resubmitted on May 12, 2009; accepted on May 23, 2009.


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